By S C Bishop; C.A.B. International.; et al
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Additional info for Breeding for disease resistance in farm animals
In 1908 Ronald Ross translated the problem from a discrete-time model to a continuous-time framework in his work on the dynamics of malaria. Subsequently, much of the theory used to develop epidemic models has been based on differential calculus used to describe rates of transmission or processes, or based on stochastic approximations to such equations. It is necessary, when modelling diseases, to consider both the modelling approach and the type of pathogen being modelled. Models are generally formulated either deterministically or stochastically, although there is overlap between these categories.
B. K. (2005) TIM-4 is the ligand for TIM-1, and the TIM1-TIM-4 interaction regulates T cell proliferation. Nature Immunology 6, 455–464. MHC Sequencing Consortium (1999) Complete sequence and gene map of a human major histocompatibility complex. Nature 401, 921–923. J. K. (2002) Th1-specific cell surface protein Tim-3 regulates macrophage activation and severity of an autoimmune disease. Nature 415, 536–541. 35 Montecino-Rodriguez, E. and Dorshkind, K. (2006) New perspectives in B-1 B cell development and function.
6 Mb region encompassing 140 genes (MHC Sequencing Consortium, 1999). The MHC region is divided into three subgroups: class I and class II, separated by class III. The picture may be different in nonmammalian vertebrates. , 1999a,b). The class I and II ‘regions’ in the chicken are side by side in the genome, rather than separated by the class III region. , 2005). , 2005). In mammals, there is only limited evidence that MHC genes confer enhanced resistance against infection and that this can be selected for.
Breeding for disease resistance in farm animals by S C Bishop; C.A.B. International.; et al