By Ronald W. Estabrook and Paul Srere (Eds.)
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Extra info for Biological Cycles
The marked decrease in the plasma level of glutathione found after inhibition of glutathione synthesis in- GLUTATHIONE METABOLISM AND TRANSPORT CYCLES 39 dicates that there is normally a substantial flow of glutathione into the plasma. g, kidney) can act on the glutathione that they synthesize and translocate to the membrane-bound enzyme. The very low concentration of glutathione in the blood plasma shows that glutathione is effectively removed from the circulation. Glutathionuria induced by administration of transpeptidase inhib itors seems to provide an experimental animal model for the human disease in which there is deficiency of transpeptidase activity.
Administration of amino acids leads to effects t h a t can best be explained in terms of increased tr anspeptida tion. IV. , in the blood plasma). γ-Glutamyl transpeptidase is bound to the membranes of cells at sites t h a t are extensively involved in transport phenomena. Since γ-glutamyl transpeptidase is accessible to externally supplied sub strates, it has been concluded t h a t this enzyme is bound to the outer surface of the cell membrane. For example, suspensions of lymphoid cells can act on γ-glutamyl substrates t h a t are impermeable to the cell membrane, and disruption of such cells does not lead to increased transpeptidase activity (110).
And Cohen, P. ,Proc. NatL Acad. Sei. A. 77, 7044-7048(1980). 106. , and Cohen, P. , J . Biol. , 256,4127-4132 (1981). 107. Cohen, P. , in "The Urea Cycle" (S. Grisolia, R. Baguena, and F. ), pp. 21-38. Wiley, New York, 1976. 108. , Paik, W. , and Cohen, P. yProc. NatL Acad. Sei. A. 69, 3530-3533 (1972). 109. , in "The Urea Cycle" (S. Grisolia, R. Baguena, and F. ), pp. 181-219. Wiley, New York, 1976. 110. , in "The Urea Cycle" (S. Grisolia, R. Baguena, and F. ), pp. 221-235. Wiley, New York, 1976.
Biological Cycles by Ronald W. Estabrook and Paul Srere (Eds.)